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Tag Archives: Specific Binding of P4 and P8 to APP
Novel Approach to Prevention of Alzheimer’s Disease Explored in Laboratory (Express / PloS One)
Summary Researchers at the University of California, San Diego, have explored a different approach to mainstream research into the prevention of Alzheimer’s Disease. It is believed that Alzheimer’s Disease develops when amyloid precursor protein (APP) gets cut-up by enzymes and … Continue reading →
Posted in Animal Studies, For Doctors (mostly), For Researchers (mostly), In the News, International, Pharmacological Treatments, Quick Insights
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Tagged Aβ40, Aβ42, Alzheimer's Disease, Amyloid Beta, Amyloid Beta (Aβ42 and Aβ40), Amyloid Beta Protein, Amyloid Precursor Protein, ß-amyloid, Beta-Amyloid, Binding Affinity Measurement by Biolayer Interferometry, Candidate Drugs for Alzheimer's Disease, Confocal Microscopy, Cultured Jurkat Cells, Department of Biology: University of California San Diego, Department of Medicine: University of California San Diego, Department of Neuroscience Imaging Core: University of California San Diego, Department of Neurosciences: University of California-San Diego, Disease Modifying Treatment by 2025 (Aim), Experimental Therapeutic Approaches to Reduce ß-Amyloid (Alzheimer’s Disease), Express (Newspaper), FortéBio, Genetically Engineered Mice, Human Neuroblastoma Cell-Line IMR-32 (ATCC CCL-127), β-Amyloid (Aβ) Accumulation, β-Amyloid Precursor Protein (APP), Mouse Model of Alzheimer's Disease, Notch Intracellular Domain (NICD), P4 and P8: Protein Site-Specific Small Peptides to Reduce Aβ Production, Pall Corporation, Peptides of Presenilin-1, Peptides P1 Through P10, PLoS One, Presenilin, Specific Binding of P4 and P8 to APP, Transgenic Mice, United States; Cenna Biosciences Incorporated, University of California, University of California San Diego, USA
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